HYBRID EVENT: Join us in person in Boston, Massachusetts, USA or attend virtually from anywhere.
Growing evidence suggests that exposure to environmental toxins & obesogens plays a significant role in obesity development. Chemicals found in plastics, pesticides, and food packaging—such as bisphenol A (BPA) and phthalates—can disrupt endocrine function and alter fat metabolism. These substances interfere with hormone signaling, leading to increased fat storage and appetite dysregulation. Prenatal and early-life exposure to obesogens has been linked to long-term metabolic changes, predisposing individuals to obesity and related diseases. Additionally, air pollution and heavy metals contribute to systemic inflammation and insulin resistance. Reducing exposure through regulatory policies, safer consumer products, and dietary choices that minimize processed food intake is essential for mitigating their impact on obesity prevalence.
